Management of hyperlipidemia medication: Avoid two major taboos: blindly adjusting medication and arbitrarily stopping medication.

Avoid Blindly Taking or Not Taking Measures: For certain special populations (such as patients with abnormal liver or kidney function) and the elderly, due to their slower drug metabolism and excretion, they are more prone to adverse drug reactions. Therefore, doctors should pay close attention to adjusting the dosage appropriately during treatment to avoid adverse drug reactions or even drug poisoning. Liver and kidney function should be checked regularly to ensure early detection, prevention, and treatment of adverse drug reactions. Patients should not blindly reduce or increase the dosage based on the improvement or worsening of their condition.

If serum alanine aminotransferase (SGPT) is more than 3 times higher than normal, or creatine kinase (CK) is more than 5 times higher than normal, the medication must be reduced or discontinued immediately. If SGPT is more than 5 times higher than the upper limit of normal or CK is more than 10 times higher than the upper limit of normal, the medication should be discontinued immediately, and appropriate treatment should be provided if necessary. Patients who experience adverse reactions after taking medication should not endure the pain and discomfort. They must be followed up until the clinical symptoms disappear and the above-mentioned symptoms disappear, returning to normal levels.

Avoid abruptly stopping medication: Hyperlipidemia often presents with few or no symptoms, but once symptoms appear, treatment becomes extremely difficult.

People often think that blood lipids only refer to triglycerides, but they also include cholesterol. Cholesterol plays a more significant role in the development of cardiovascular and cerebrovascular diseases. Cholesterol is mainly found in low-density lipoprotein (LDL), which acts like silt in a pond, entering blood vessels and adhering to the vessel walls to form atherosclerotic plaques, causing narrowing or blockage of blood vessels. Even more frightening is that the "fibrous cap" on the surface of these plaques can rupture, causing the plaque contents to react with the blood, forming a thrombus in a short time. This can suddenly block the major blood vessels in the heart (coronary arteries), leading to sudden death without any symptoms.